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Trestolone

Also known as: MENT, Trestolone Acetate, 7α-Methyl-19-nortestosterone

10
1 · Natty510 · Enhanced

Lower numbers = closer to natural. Higher numbers = more enhanced.

Why this rating?

MENT has been documented since 1960s bioassay research as roughly 10x the anabolic potency of testosterone per milligram, making it one of the most potent androgens ever characterized (WADA scientific-research summary on trestolone metabolism). It was developed and clinically trialed specifically as a male contraceptive because even single-digit milligram doses profoundly suppress LH, FSH and endogenous testosterone (Suvisaari et al. 1997, PMID 9194649, which tested daily IM doses of only 1-4mg), subdermal implant trials tested for hormone replacement delivered even less, on the order of well under 1.5mg/day. Enhancement doses run an order of magnitude above that, driving dramatic muscle growth far beyond any natural capacity and placing MENT at the top of the scale alongside trenbolone and methyltrienolone.

Overview

A synthetic androgen 10 times more potent than testosterone, initially developed for male contraception. Can serve as a replacement for testosterone as a cycle base due to its conversion to estrogen (unlike other 19-nors).

Important Warnings

  • Novel compound with limited long-term human data
  • Extremely suppressive - was developed as male contraceptive
  • Aggressive estrogen management required
  • Gyno risk comes from both estrogen and progesterone, an AI alone may not cover it
  • Finasteride/dutasteride do NOT protect the hairline (not a 5-AR substrate)

Purpose & Use Cases

Extreme Hypertrophy

Provides dramatic muscle growth at lower doses than traditional steroids.

Testosterone Replacement

Can serve as the sole hormonal base in a cycle, unlike other 19-nors.

Rapid Results

Short ester provides quick saturation and visible results.

Benefits

  • Extreme anabolic potency (10x testosterone)
  • Can replace testosterone as base
  • Rapid results with acetate ester
  • Significant muscle fullness
  • Maintains libido and sexual function (unlike Deca/Tren)
  • Provides estrogen (no need for exogenous estrogen)

Good to Know

The one 19-nor that can stand alone

Unlike Deca and Tren, MENT aromatizes to its own estrogen, so it can be run as the sole hormonal base and generally preserves libido rather than causing "deca dick." That is its defining feature, but it means you must manage estrogen actively, because nothing else is supplying it.

Two gyno pathways: estrogen AND progesterone

MENT is both strongly aromatizing and progestogenic, so gyno risk comes from two directions. Keep an AI for the estrogen and be ready to control prolactin (cabergoline/P5P) for the progestin side.

Not 5-alpha-reduced: finasteride is useless

The 7-alpha-methyl group blocks 5-alpha-reductase, so finasteride/dutasteride cannot help the hairline. Any hair loss is via direct androgen action, with no 5-AR step to block.

Contraceptive-grade suppression and thin human data

MENT was developed as a male contraceptive precisely because it shuts the axis down hard. Recovery needs aggressive PCT. It is a novel compound with limited long-term human data, and its ~10x potency with dramatic growth even at low doses is why it rates a 10. Muscle memory means retained gains keep your natural ceiling higher afterward.

Its self-made estrogen isn't a full substitute long-term

In hypogonadal-replacement trials, MENT matched testosterone for libido, mood and sexual function, but researchers described its own estrogenic activity as comparatively weak and observed decreased bone mineral density with extended use, meaning the estrogen MENT itself supplies may not fully protect bone health over long continuous use. This is separate from (and does not reduce) the real gyno/water-retention risk seen at bodybuilding-level doses.

Dosage Guidelines

Experience LevelDosage Range
Beginner1025 mg/day
Intermediate2535 mg/day
Advanced3550 mg/day
Frequency
Daily injection (subcutaneous or IM) due to very short half-life
Typical Cycle Length
48 weeks
Notes

Doses are much lower than traditional steroids due to extreme potency. NOTE: these ranges are community/harm-reduction-derived from enhancement forums, not clinical data, clinical trials only ever tested hormone-replacement-level exposure (roughly 1-4mg/day via injection, or well under 1.5mg/day via subdermal implants), with no bodybuilding-dose literature to draw on. Because both the parent compound and the acetate ester clear within hours, steady blood levels are reached quickly with daily dosing. There is no meaningful "loading phase" the way there is with long esters. Some experienced users report pushing 50-75mg/day, but other accounts describe deliberately capping well below that after struggling to control estrogenic/progestogenic side effects, treat the top of any range as high-risk, not a target.

Half-Life

Very short and imprecise: commonly cited at roughly 4-12 hours for trestolone and its acetate ester (estimates vary by source); requires daily injections to maintain stable blood levels.

Side Effects

Severe HPTA Suppression

very common
Severity
5/5

Extremely suppressive, originally developed as male contraceptive.

Mitigation

Aggressive PCT required; HCG during cycle may help.

Aggressive Aromatization

very common
Severity
4/5

Aromatizes heavily, requiring vigilant estrogen management.

Mitigation

AI required (Arimidex/Letrozole). Start low and titrate based on symptoms/bloodwork.

Hair Loss

common
Severity
3/5

Can accelerate hair loss despite not converting to DHT.

Mitigation

Limited options; RU58841 may provide some protection.

Gynecomastia

common
Severity
4/5

High gyno risk due to both estrogen and possible progestogenic activity.

Mitigation

Keep AI on hand; monitor for early signs.

General Mitigation Strategies

Aggressive estrogen management with Arimidex or Letrozole. Start with lower doses to assess aromatization. Regular bloodwork essential due to novel nature of compound.

Support Supplements

Ancillary supplements commonly run alongside Trestolone to manage side effects, support the target tissue, or fill nutrient demands it creates.

Aromatase inhibitor (anastrozole / exemestane)

Key

MENT aromatizes to 7-alpha-methylestradiol, a potent estrogen, so estrogenic gyno/water risk is real and vigilant AI use is genuinely needed here.

Dose
Anastrozole 0.25-0.5mg EOD, dosed to bloodwork
Timing
Through the cycle
When
Titrate to E2 symptoms/labs: because it is the sole base, crashing estrogen leaves you with nothing else supplying it.

Cabergoline (or P5P) for prolactin

As a 19-nor progestin, MENT can raise prolactin and add a progesterone-driven gyno pathway on top of the estrogenic one. Cabergoline controls it; P5P is a mild alternative.

Dose
Cabergoline 0.25mg 2x/week; P5P 100-200mg/day
Timing
Dosed to prolactin bloodwork

Lipid + blood pressure support (omega-3 / citrus bergamot / telmisartan)

A potent androgen that suppresses lipids and can raise blood pressure; standard cardiovascular support applies.

Dose
Omega-3 2-4 g/day; citrus bergamot 500-1,000 mg/day; telmisartan 20-40 mg/day if BP is high
Timing
With meals; BP meds to readings

Post Cycle Therapy (PCT)

⚠️PCT Required

Aggressive PCT required due to extreme suppression. Due to short half-life of acetate, can begin PCT within days.

How It Works

MENT is a 19-nor testosterone derivative that is not 5α-reduced to DHT but does aromatize to estrogen. It has approximately 10x the anabolic potency of testosterone and can maintain normal physiological function without a testosterone base.

Hormonal & Androgenic Profile

Aromatizes (→ estrogen)high
Natural test suppressiontotal
Hair loss risk (DHT-prone)moderate
Liver toxicitynone
DHT-derivativeNo
Progestogenic (19-nor)Yes
Anabolic : Androgenic ratio

Roughly 10x the anabolic potency of testosterone (7-alpha-methyl-19-nortestosterone), one of the most potent androgens known

Estrogen control

Aromatizes to 7-alpha-methylestradiol: vigilant AI use (anastrozole/exemestane) is genuinely needed at enhancement doses. Because it makes its own estrogen, MENT can serve as a standalone base without added testosterone, unlike other 19-nors; note that hormone-replacement studies described this self-supplied estrogenic activity as comparatively weak per milligram versus testosterone (Wikipedia summary of clinical literature), so at replacement-level doses it under-delivers estrogen, while at supraphysiological enhancement doses the absolute estrogen load is enough to require real AI management.

DHT / 5-AR & finasteride

MENT is not 5-alpha-reduced (the 7-alpha-methyl group blocks it), so finasteride/dutasteride do nothing. It can still thin hair via direct androgen action, but there is no 5-AR conversion to block.

Cardiovascular impact

Suppresses lipids and can raise blood pressure like other strong androgens; estrogenic water retention is possible if E2 runs high. Long-term human safety data are limited.

Fundamentals

WADA Status

Prohibited by WADA
Category: S1. Anabolic Agents
In-Competition: ProhibitedOut-of-Competition: Prohibited

MENT (7α-methyl-19-nortestosterone) is prohibited as an anabolic androgenic steroid.

References

Last updated: July 18, 2026